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Display Strains By Phenotype


 

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adipose tissue lethality-embryonic/perinatal
behavior/neurological lethality-postnatal
cardiovascular system life span-post-weaning/aging
cellular phenotype limbs/digits/tail phenotype
craniofacial liver/biliary system
digestive/alimentary muscle
embryogenesis nervous system
endocrine/exocrine gland no phenotypic analysis
growth/size normal phenotype
hearing/vestibular/ear other phenotype
hematopoietic pigmentation
system homeostasis/metabolism renal/urinary system
immune system reproductive system
     adenovirus infection of macrophages- decreased respiratory system
     adenovirus infection of macrophages- increased skeleton phenotype
     B cell deficiency skin/coat/nails
     CD4 deficiency taste/olfaction
     CD8 deficiency touch/vibrissae
     decreased CD8 response tumorigenesis
     decreased NK cell response vision/eye
     decreased response to injected CpG DNA  
     impaired response to dsDNA- type I IFN production by macrophages  
     Inflammatory bowel disease phenotype  
     influenza proliferation in macrophages- decreased  
     influenza proliferation in macrophages- increased  
     Influenza resistance  
     MCMV proliferation in macrophages- decreased  
     MCMV proliferation in macrophages- increased  
     MCMV resistance  
     MCMV susceptibility  
     NALP3 inflammasome signaling defect  
     NK cell deficiency  
     NKT cell deficiency  
     RVFV proliferation in macrophages- decreased  
     RVFV proliferation in macrophages- increased  
     RVFV susceptibility  
     T-dependent humoral response defect- decreased antibody response to rSFV  
     T-dependent humoral response defect- increased antibody response to rSFV  
     T-independent B cell response defect- decreased TNP-specific IgM to TNP-Ficoll immunization  
     TLR signaling defect- hypersensitivity to CpG  
     TLR signaling defect- hypersensitivity to LPS  
     TLR signaling defect- TNF production by macrophages  
     TLR signaling defect- type I IFN production by macrophages  
     TLR signaling defect- type I IFN production by pDC